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Such approaches enable the synchronized modulation of inflammation, proliferation, and tissue remodeling through bioresponsive physicochemical cues, representing a paradigm shift toward actively participating biomaterials [4]
Third, inhibition of c-Met signaling pathways triggers the EGFR pathway as a compensatory survival pathway (Steinway et al., 2015)
This flexibility in dosing options allows healthcare providers to customize treatment regimens based on individual patient responses, tolerability, and therapeutic goals
10.1142/S0192415X23500209 5 BingL
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