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Although there is literature based on investigating the cross-sectional association between UPFs and mental disorders, there continues to be a relative lack of high-quality evidence on the prospective effects of UPFs as a risk factor for the development of mental disorders, as well as the pathophysiological mechanisms involved (21, 22)

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In 1990, Frosst et al (28) identified a C677T polymorphism in the MTHFR gene that reduced enzyme activity and altered folate concentrations

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