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Karbalay-Doust S et al

Mechanisms include: Restoration of endothelial nitric oxide synthase (eNOS) Upregulation of VEGF and FGF-2 (fibroblast growth factor 2) Reduction of oxidative stress and cytokine-induced apoptosis BPC-157 preserved endothelium-dependent vasodilation via upregulation of eNOS and suppression of vascular inflammation. Gwyer Findlay et al., Peptides 4

Chakrabarti, P., English, T., Shi, J., Smas, C

High enough to be bacteriostatic (inhibiting bacterial growth) without being bactericidal (killing bacteria outright at first contact)

In summary, after consideration of the public comment, we are finalizing our proposal without modification