The default systemic-repair peptide
Ornithine (an arginine byproduct) suppressed the metabolism of CD8 + T cells, reducing the intracellular levels of adenosine triphosphate (ATP), and, at the same time, increasing the resistance of tumor cells to CD8 + T cells-mediated cytotoxicity [87]
In mice, DA is degraded in the caudate nucleus via the DAT pathway within 200 milliseconds, in comparison with 2,000 milliseconds in the frontal cortex [11]
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