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Furthermore, the interplay of NO and H 2 S in vascular modulation, particularly through agents such as SNP and Na 2 S, offers potential in regulating endothelial dysfunction and oxidative stress

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The accumulation of -Syn in excess also activates the autophagy-lysosomal pathway (Zhang et al., 2018), mutations in -Syn proteins undergo misfolding in transcription-translation to produce toxicity, and defective mitophagy further exacerbates neurotoxicity, leading to neuronal loss or damage (Wang et al., 2018)