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These models allow the investigation of molecular pathways underlying tumorigenesis and cellular processes driving nodule formation, providing critical insights into the pathophysiology of thyroid cancer
However, when the physiological demand for antioxidants surpasses their bioavailability, the brain enters a state of REDOX imbalance in which ROS accumulate, leading to neuronal damage [6, 7]
DOI: 10.1080/07315724.1990.10720349
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