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Imatinib, the iron-chelating agent deferoxamine, and lactoferrin all showed protective effects in ferroptosis-induced cardiac dysfunction induced by COVID-19 [133, 134]
It acts as both a GLP-1 and a glucose-dependent insulinotropic polypeptide (GIP) receptor agonist
This function is particularly relevant in neurons, where sHSPs frequently colocalize with disease-associated protein aggregates and serve as an early line of defense against proteotoxic stress
Assignment of other roles, beyond the anti-oxidative ones, to the GSH enhancement in the resistance to FLC should be considered more speculative
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