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Zhang, Q., Yan, L., Lu, J., & Zhou, X

This signaling pathway is an adaptive mechanism that activates the expression of nuclear genes such as NF-KB by transmitting dysfunctional mitochondrial signals [114], thereby triggering neuroinflammation and cognitive impairment

However, the absence of Hippo signaling triggers unphosphorylated YAP/TAZ to translocate into the nucleus and activate mainly the transcriptional enhanced associate domain (TEAD) family of transcription factors to drive Hippo target gene expression in the liver

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