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Hypocalcemia can trigger secondary hyperparathyroidism, where the parathyroid glands become overactive in an effort to stabilize blood calcium levels

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Furthermore, mice made diabetic by streptozotocin (STZ) administration, but genetically deficient for the GcgR, display normal glucose tolerance [49]

Rojstaczer, N

Cell viability for GHK-Cu was previously investigated by Hairui et al, showing no significance difference between the control and the GHK-Cu treated group at the same concentrations and time points, implying the non-cytotoxic nature of GHK-Cu towards HaCaT 11