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Results: We found that CB-839 caused dose-dependent decreases in GLS-1 activity and glutathione synthesis

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Studies by Pickart and colleagues published in 2012-2018 showed that GHK-Cu treatment of cultured human fibroblasts altered expression of several thousand genes, with patterns indicating increased expression of genes involved in tissue repair, antioxidant responses, protein synthesis, and cellular rejuvenation

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These include neurodegenerative disorders, liver diseases, and inflammatory conditions because of their ability to scavenge free radicals and reduce oxidative damage