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Various mechanisms have been theorized to describe how dual agonism, as GLP-1 RA, could directly reduce triglycerides hepatocyte storage, lipogenesis and improving hepatic glucose metabolism (96) and promoting lipolysis and fatty acid oxidation (97)

Most people who use GLP-1 medications do not develop gastrointestinal paralysis

In fact it runs in fatty acids, it runs on other things like lactate and ketones, but it loves fatty acids

The subsequent increase in nitric oxide (NO) production is a critical mediator of vasodilation and vascular permeability, processes essential for tissue repair

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