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Furthermore, DPP-4i could inhibit the expression of MCP-1 in mouse proximal TECs upon free fatty acid binding to albumin and significantly attenuate renal tubulointerstitial injury (172)

We were thus prompted to explore the intriguing hypothesis that GSH-dependent control of ROS could play a role in defining the distinctive metabolic signaling of MZB and FoB

Hypertens Preg 43:2404459

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