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Mutations in superoxide dismutase 1 (Sod1) linked to familial amyotrophic lateral sclerosis can disrupt high-affinity zinc-binding promoted by the copper chaperone for Sod1 (ccs)

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This raises the possibility that if a patient ingests some iron supplement tablets during a suicide attempt with an overdose of APAP [83, 84], the mechanism of cell death is switched to ferroptosis, and the accelerated and enhanced severe toxicity can be rapidly fatal [85]