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In a subsequent study using the protein toxin CNF-1 to increase the activity of Rho proteins, it was found that CNF-1 treated mice had an elevated level of actin filaments and enhanced freezing during a fear memory test and better performance in the Morris water maze test (Munro et al., 2004)

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In preclinical models of traumatic brain injury (TBI) or sciatic nerve crush injury, investigations focus on the potential to reduce neuronal apoptosis, mitigate inflammatory responses, and promote axonal regeneration

Dominant psychiatric manifestations in Wilsons disease: a diagnostic and therapeutic challenge