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When addressing severe oxidative stress or toxic exposure recovery, temporarily elevated dosages may reach 2000-3000mg daily for limited periods under professional guidance

These results corroborated previous findings of Harris et al

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Observed mechanisms include direct neuronal damage, disruption of key neurotransmitter pathways such as AChE activity, induction of cellular stress responses, and alterations in both locomotor and spontaneous activity patterns, particularly in Drosophila