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At therapeutic doses of tretinoin, the activity of the fusion protein on differentiation converts from inhibitory to stimulatory

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G6PD deficiency causes increased susceptibility of erythrocytes to H2O2 and other reactive oxygen species that can lead to haemolytic anemia, favism, chronic non-spherocytic hemolysis, spontaneous abortions, and neonatal hyperbilirubinemia resulting in neonatal kernicterus

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At the same time, studies could also aim to validate the associations between these SNPs and micronutrient levels in the general population to ensure their consistency across different populations