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The GHS receptor was renamed the ghrelin receptor (GHS-R1a), and the synthetic GH secretagogues were understood to be mimicking a gut-brain signaling axis that linked nutritional status to growth hormone release

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The glycine effect on insulin resistance in SF rats can also be related to its decreased effect on liver free fatty acid level which is well known to induce lipotoxicity and promote ROS generation in excess and mitochondrial dysfunction [17]

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