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All together, these data suggest a crucial role of CD36 in PA-induced ferroptotic cell death in TNBC through the increase of different hallmarks of ferroptosis, especially peroxidized lipid, mitochondrial ROS accumulation, and mitochondria morphology, as well as increased iron uptake leading to an increase Fe 2+ content
Cytoprotection by almond skin extracts or catechins of hepatocyte cytotoxicity induced by hydroperoxide (oxidative stress model) versus glyoxal or methylglyoxal (carbonylation model) Chemico-Biological Interactions
