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[13] At the hypothalamic-pituitary axis, estradiol down-regulates kisspeptin neuron activity, suppressing gonadotropin-releasing hormone pulsatility and thereby reducing luteinizing and follicle-stimulating hormone secretion-actions that are largely clinically silent in post-menopausal patients but relevant in pre-menopausal users

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In this context, SCMC-related signatures should be interpreted as coordinated programs governing oocyte-to-embryo continuity rather than as isolated markers