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Increased reliance on fatty acid oxidation (FAO) alters mitochondrial electron flux and redox cofactor dynamics, influencing both anaboliccatabolic balance and oxidative buffering capacity

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93 Endogenous antioxidant enzymes such as SOD have been shown to protect the brain by reducing apoptosis in mouse models by suppressing the activation of extracellular signal-regulated kinases 1 and 2

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