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Arginine metabolism plays a double-edged role in regulating macrophage function: on the one hand, arginine is metabolized by iNOS into NO to promote M1 responses

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Although reactive electrophiles such as chloroacetamides are generally considered to be promiscuous in target binding, we demonstrated here using a BODIPY-labelled analogue that the chloroacetamide-based GSTO1 inhibitor C1-27 exhibited a low degree of nonspecific reactivity

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