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ASA treatment at 5 mol/ml caused only 1015% inhibition in cell viability while 10 mol/ml ASA caused about 3040% loss in cell viability in 48 hours [23]

GLP-1 receptor agonist inhibited proinflammatory monocyte chemoattractant protein-1 secretion from human atherosclerotic plaque samples and monocyte chemoattractant protein-1 (MCP-1), tumor necrosis factor- (TNF-), and interleukin (IL)-1 secretion from human macrophages after ex vivo treatment

These typically resolve without intervention and do not necessarily indicate hepatotoxicity