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The precise origin of non-mitochondrial CoQ utilized by FSP1 for ferroptosis defense requires further investigation

Smilansky and colleagues suggested that neurodegeneration in AD could be ameliorated by downregulation of overexpressed VDAC1 RNA levels by small interfering RNA (siRNA) or by the use of VDAC1 Nterminal peptide (VDAC1Nter) [223]