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Elevated cortisol directly inhibits GH receptor signaling in target tissues and increases hepatic GH resistance, meaning even if you achieve a GH pulse, downstream IGF-1 production and metabolic signaling are impaired

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Proliferative cells have more negative steaty-state redox potential and differentiated cells have more positive cells and cells undergoing apoptosis and necrosis have a more oxidized steady-state potential (around -170 mmol/L to -150 mmol/L)[45]