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To restore NAD + levels, ATM-deficient models were treated with NR, which stimulated PINK1-dependent mitophagy, reduced cytoplasmic DNA levels, and activated the NAD + -dependent SIRT1 signaling pathway, which in turn further promoted mitochondrial mitophagy and healthy morphology 111,288

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These droplets also harden and lose their dynamic properties

267 Although these trials utilized combination therapy rather than sequential therapy, strategies combining senescence inducers (e.g., etoposide, olaparib, or paclitaxel) with senolytics (e.g., navitoclax) have demonstrated clinical benefits in studies