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In murine models treated with the neurotoxin MPTP, which selectively targets dopaminergic neurons to simulate PD, semaglutide administration effectively safeguarded these neurons from apoptosis, maintained dopamine concentrations, and subsequently enhanced motor function ( 4.3 Cerebrovascular disease models Given the established association between vascular health and cognitive function, the effects of semaglutide on stroke models are of particular significance

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