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Furthermore, caveolin 1 knockout augmented steatosis both in vitro and in vivo by enhancing the expression of genes involved in DNL [30], contradicting earlier studies, and suggesting a protective effect of caveolin 1 in NAFLD [30]

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458 Inflammatory cytokines and autoantibodies implicated in RA pathogenesis can activate ER stress pathways, which in turn drive inflammatory responses that contribute to disease progression