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Operating as a ligand for IGF1R, LR3 IGF-I binds to the alpha subunit, initiating the activation of intrinsic tyrosine kinase activity, autophosphorylating tyrosine residues in the beta subunit

J Exp Med (2005) 201:23340

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Sulforaphane treatment of diabetic mice markedly restored Nrf2 expression, increased the levels of its downstream target proteins related to antioxidative defense, reversed the increase in iron accumulation by suppressing the colocalization of FTH1 and LC3 for degradation and by diminishing iron absorption from endosomes, and finally prevented iron-induced lipid peroxidation and ferroptosis (90)