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2-month treatment with exenatide in mice promotes the enhancement of long-term memory performances along with the activation of the BDNF-TrkB neurotrophic axis and inhibits apoptosis by decreasing p75NTR-mediated signaling [50, 51]

Medications distribute differently across these compartments, and the rate of distribution affects the concentration curve

Neuroprotection and its molecular mechanism following spinal cord injury

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These findings are in agreement with the results of a clinical trial by Duranay, et al