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Understanding the differences between Phase I and Phase II metabolism is crucial for assessing the overall metabolic fate of drugs and xenobiotics, predicting their pharmacological activities and toxicity, and optimizing drug therapy

For example, the p53 mutants p53 R 237 H and p53 R 175 H promote sensitivity to ferroptosis-like cell death, most likely through the combination of p53 mutants with NRF2 and through the suppression of the NRF2-dependent transactivation of SLC7A11 together with other antioxidant genes that oppose ferroptosis (Sasaki et al., 2002

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Phytochemical investigation and radical scavenging activities of Melia azedarach and its DNA protective effect in cultured lymphocytes

The intestinal flora also plays an important role in other PPI-mediated diseases