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The aim of this study is to exploit GSH-responsive AP site capture reagent (AP probe-net), which responses to the elevated glutathione (GSH) levels in the tumor micro-environment (TME), releasing reactive alkoxyamine to trap AP sites and block the APE1-mediated BER for targeted anti-tumor activity against ATC

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Patients with a history of medullary thyroid carcinoma, pancreatitis, or severe gastrointestinal disease should avoid these medications

Glucagon-Like-Peptide 1 (GLP-1)