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A key role for NOX4 in epithelial cell death during development of lung fibrosis
287,288 This binding initiates typical GPCR signaling, leading to G protein activation and increased intracellular cAMP
These findings suggest that manoeuvres designed to increase mucosal GSH content may be useful to withstand the lethal consequences of oxidative stress that may contribute to attenuate the activation and recruitment of related factors that participate in the development of IBD, including VCAM-1 and ICAM-1 (Sans et al, 1999)
2006 Sep;16(9):688-91
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