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Its initiation depends on intracellular iron overload, which serves as the key biochemical basis and trigger, ultimately leading to extensive membrane damage

Like virtually all medications, incretin mimetics come with side-effects, leading some patients to abandon treatment

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found that ferroptosis in ectopic endometrial stromal cells in patients with ovarian endometriosis could activate the p38 mitogen-activated protein kinase (p38 MAPK)/signal transducer and activator of transcription (STAT) 6 signalling pathway, thereby promoting local upregulation of vascular endothelial growth factor A (VEGFA) and interleukin-8 (IL-8) in ectopic lesions [15]

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