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Solubility 10 mg/mL in sterile PBS (pH 7.4)

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By looking at individual cells, scientists gain new, more precise information on the genetic differences between the brain of someone with a disease compared to someone without the disease

Future work will use longitudinal profiling and single-cell sequencing to assess cell-type-specific GSH roles 5 Conclusion In conclusion, by leveraging multi-omics integration, the present study identified GSH metabolism as a pivotal node in SAE, while providing a comprehensive molecular framework for SAE pathogenesis, and linking microglia-driven neuroinflammation to hippocampal oxidative damage