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Identifying genes and proteins specifically associated with tumor traits would facilitate the development of therapies that selectively target tumor cells while minimizing toxic effects

Potent anti-fibrotic agent SalB suppressed autophagosome formation and autophagic flux in HSC by down-regulating the extracellular regulated protein kinases (ERK), c-Jun N-terminal kinase (JNK), and p38-MAPK pathways, leading to decreased expression of light chain 3 II, autophagy-related gene 5 (Atg5), -SMA, and CoI ( SalB might be a useful and potent TGF- antagonist to postpone the development of HF

Translational control of inducible nitric oxide synthase expression by arginine can explain the arginine paradox

MagrinelliFPicelliAToccoPFedericoARoncariLSmaniaNet al

In most cases, when the trigger medication is discontinued, abnormal liver tests reverse to normal