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Deficiency in the level of activity of G6PDH is the basis of favism, primaquine (an antimalarial drug) sensitivity, and several other drug-sensitive hemolytic anemias, anemia and jaundice in the newborn, and chronic nonspherocytic hemolytic anemia

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It also diminished induction of TNF- and IL-6, decreased NF-B nuclear translocation and p38 mitogen activated protein kinase phosphorylation, abolished intracellular hydrogen peroxide production, restored intracellular thiol levels, and reduced virus titre