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[2] Treated animals showed progressive recovery of tail motor function, absence of autotomy, and resolution of spasticity, alongside electromyographic changes and a microscopic profile in which the vacuolization, white-matter axon loss, gray-matter edema, and motoneuron loss typical of the injury were attenuated

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Additionally, it has been discovered that TAZ serves as a genetic determinant of ferroptosis through the regulation of ANGPTL4-NOX2, thereby highlighting its potential as a novel therapeutic target for ovarian cancer and other TAZ-activated tumors (Yang et al., 2020)