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Mechanistically distinct from other forms of cell death, cuproptosis is characterized by the direct binding of copper to lipoylated enzymes in the mitochondrial tricarboxylic acid (TCA) cycle, leading to proteotoxic stress and subsequent cell death, independent of classical apoptotic signaling [3]

Association of three genetic loci with uric acid concentration and risk of gout: a genome-wide association study

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Setria Glutathione Studies 5b