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identified mitophagy as an intrinsic vulnerability in CRC cells subjected to statin treatment, revealing how disruption of mitochondrial homeostasis, shaped by distinct metabolic contexts, leads to cell death and therapeutic sensitivity [103] Spatial heterogeneity across colorectal tumor sites underlies distinct metabolic phenotypes [104]

Cationic, anionic, neutral, and slightly anionic NPs have been reported to be cleared from fastest to slowest, respectively [36]

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These criticisms stem from concerns about curcumins interference with multiple research assays, complicating the assessment of its true biological activity