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Thus, these in vitro findings indicate that the beneficial effect of incretin receptor agonists on fibrosis progression in MASLD patients is likely to be mediated indirectly through improvements in body weight, inflammation, and insulin resistance, rather than through the direct action on hepatic parenchymal or non-parenchymal cells

Bakn, glutatyon bizi korumay nasl baaryor

Unknown risk is not the same as low risk

The different mutations in SLC22A5 probably give rise to differences in severity/onset of the disease

Moreover, most PDMS-based OoC rely on irreversible closing systems, impeding the opening of the chip before endpoint analysis, thus limiting sample accessibility and post-culture interventions (78)