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Proteins downregulated by GLP-1GIPLani (cluster 1) were mainly associated with calcium and tubulin binding, ion channel and phosphatase regulation and protein folding, whereas proteins upregulated by GLP-1GIPLani (cluster 3) were linked to oxidative and lipid metabolism, ribosomal activity and amino acid and carbohydrate metabolism (Fig

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This paradox underscores the complexity of FASNs role in EC biology, suggesting that both excessive and insufficient DNL can contribute to endothelial dysfunction, depending on the context and temporal dynamics