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Upon binding to its own receptor, IGF-1 signals the pituitary gland to secrete less growth hormone and, at the same time, tells the hypothalamus to secrete less GH-releasing hormone

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Protection of the BBB, the inhibition of the elements of the brain microvascular damage such as impairment of tight junctions, increased vesicular transcytosis, production of various cytokines, chemokines and reactive oxygen species offers novel therapeutic targets [11]