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These effects were reversed by ferrostatin-1, a specific inhibitor of ferroptosis, confirming the ferroptosis-dependent nature of NO-induced cell death in CRC models ( This review examines NOs role in CRC progression, highlighting its effects on molecular pathways, immune modulation, and metabolism

This has led researchers to explore whether the pair may produce complementary or synergistic signaling responses in selected in-vitro and in-vivo systems

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