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Despite the hype, liposomal and intravenous NAD+ products also fall short, as they do not reliably replenish cellular NAD+ levels

Wang L, Song YY, Wang Y, Liu XX, Yin YL, Gao S, et al

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In addition, exendin-4 treatment reduced lipid accumulation in human primary monocytes and THP-1-derived macrophages stimulated by ox-LDL [15]

The practical implication is that GHK-Cu addresses structural skin aging (collagen loss, elasticity decline) while neurotransmitter-modulating peptides address dynamic wrinkles (expression lines)