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Thus, we have demonstrated that GSH-targeted therapy selectively kills PC cells in an oxidative stress-dependent manner and that the differences in basal ROS level and antioxidant scavenging capacity induced by stratified expression of VRK2 can well account for the heterogeneous sensitivity to GSH inhibition

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A treatment with reduced DTT increased activity of the previously defined class B ATPS, which are characterized by the presence of five conserved cysteines, but not of ATPS from the A class despite they also contain conserved cysteine residues but at different positions (Prioretti et al., 2014, 2016)

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