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Of these 32 semaglutide-treated patients that developed anti-semaglutide antibodies, 19 patients (0.6% of the overall population) developed antibodies cross-reacting with native GLP-1

The compounds operate through distinct, non-overlapping mechanisms (GLP-1/GIP receptor agonism versus NNMT enzyme inhibition) with no documented receptor competition or adverse pharmacokinetic interactions

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