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Such interactions create crosstalk between epigenetic layers: MeCP2, a methyl-DNA binding protein, can recruit histone modifiers, while histone acetylation status influences DNA methyltransferase access to chromatin 181,182

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The rate-limiting reaction mechanism is depicted in Figure 3

Through division of labor, cooperation, and multi-point coordination with multi-level functional encoding, they can navigate complex environments and inhibit tumors via multiple pathways ( Substantial interdisciplinary progress is emerging