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Aberrant K63-linked ubiquitination disrupts intracellular signaling networks and autophagic flux, contributing to the accumulation of toxic protein species and impaired stress responses

stvold at the Department of Clinical Dentistry, Liv Kristine ysd and Torunn Eide, Department of Clinical Science, University of Bergen, for technical assistance

Research Interest in GHK-Cu GHK-Cu research appears throughout scientific literature involving copper peptides, gene expression research, extracellular matrix biology, molecular signaling pathways, peptide chemistry, cellular communication, receptor biology, and analytical peptide characterization

Experimental validation Dose response curves and determination of IC 50 values were assessed for the 12 candidate drugs using 3-fold dilution series (see Supplementary Methods (Supplementary File 1) for more details)

This study aimed to investigate the therapeutic effect of exogenous GHK-Cu on UC mice and explore its mechanism