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These alterations that are associated with VPA and trichostatin A appear to be caused by HDAC inhibition

Across preclinical studies, Epitalon has shown: Telomerase activation and telomere extension: Via TRAP assays and fibroblast lifespan extension beyond the Hayflick limit Epigenetic remodeling: Reversal of chromatin aging, histone modulation, and gene promoter binding Redox homeostasis: Increased antioxidant enzymes (SOD, NQO1, catalase) and reduced ROS, lipid peroxidation, and DNA damage Circadian restoration: Upregulation of AANAT/pCREB, normalized melatonin and cortisol rhythms, and clock gene modulation in humans Cancer risk reduction: Reduced tumor multiplicity and delayed onset in HER-2/neu and colon cancer models

In: Avers D, Wong RA, editors

These are often dose-related and may improve with slower dose escalation, hydration, fiber, protein planning, and individualized adjustments

3,28 Hepatic lipotoxicity may occur because the accumulated liver lipids stimulate the secretion and increase the activity of IL-11 protein, which upregulate NOX4 expression to increase the production of reactive oxygen species