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Single-peptide therapy often supports one part of that network well, then stalls when other constraints take over

We also don't use peptides in place of standard medical care for a diagnosed condition

The elevated calcium levels also impair mitochondrial function, triggering the release of proapoptotic factors, including cytochrome c, and activating caspase-dependent apoptotic pathways ( tThe excitotoxicity induced by METH also triggers neuroinflammation and amplifies its neurotoxic effects

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Proc Natl Acad Sci U S A 101: 12001205